Vx gas effects?

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Abraham Mante asked a question: Vx gas effects?
Asked By: Abraham Mante
Date created: Wed, Mar 10, 2021 2:32 AM
Date updated: Fri, Nov 25, 2022 2:12 AM

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Video answer: What is vx nerve agent?

What is vx nerve agent?

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Individuals who are exposed to a low or moderate dose of VX Gas by inhalation, ingestion, or skin absorption may experience some or all of the following symptoms within seconds to a few hours after exposure: runny nose, watery eyes, small pupils, eye pain, blurred vision, drooling and excessive sweating, cough, chest ...

People exposed to a low or moderate dose of VX by inhalation, ingestion (swallowing), or skin absorption may experience some or all of the following symptoms within seconds to hours of exposure: Abnormally low or high blood pressure. Blurred vision. Chest tightness.

Video answer: Health effects of nerve agents 1963

Health effects of nerve agents 1963

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The VX nerve agent causes nerves to become dysfunctional and for muscles to go into spasm. This can affect the nerves and muscles throughout the body. However, in terms of its lethal effects, it is the disruption of the nerves responsible for breathing and the spasm of the muscles of the airways and respiration that are most significant.

Early symptoms of exposure to VX vapor include rhinorrhea (runny nose) and/or tightness in the chest with shortness of breath (bronchial constriction). Miosis (pinpointing of the pupils) may be an early sign of agent exposure, but is not usually used as the only indicator of exposure.

Exposure to a large dose of VX by any route may result in these additional health effects: Convulsions ; Loss of consciousness ; Paralysis ; Respiratory failure possibly leading to death ; Showing these signs and symptoms does not necessarily mean that a person has been exposed to VX. What the long-term health effects are

Skin exposure: Liquid VX may produce health effects within minutes. Health effects from mild to moderate exposure may be delayed up to 18 hours; larger exposures may cause death within minutes to hours.

The clinical effects of VX exposure result primarily from its inhibition of acetylcholinesterase (AChE), although it does inhibit other cholinesterases as well, including butyrylcholinesterase (BuChE).

The immediate symptoms of VX nerve agent exposure include blurred vision, chest tightness, drooling and sweating, rapid breathing, and an abnormally low or fast heart rate.

VX primarily affects neurotransmitter receptors, those of norepinephrine, and also affects the central nervous system unrelated to AChE inhibition. The balance between nicotinic receptor potentiating effects and muscarinic effects at parasympathetic postganglionic fibers determines the effect of nerve gases on the heart.

Human exposure to nerve agents such as VX results in a cascade of toxic effects relative to the exposure level including ocular miosis, excessive secretions, convulsions, seizures, and death. The primary mechanism behind these overt symptoms is the disruption of cholinergic pathways.

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